Epiretinal film (ERM) is one of the best accepted retinal diseases in age-old persons,1 which after-effects in blurred eyes or metamorphopsia, such as macropsia and micropsia. Several theories that explain the mechanisms that account metamorphopsia due to ERM accommodate macular distortion, vascular leakage, edema of the close retina, and changes in the photoreceptors,2–7 but little assertive affirmation supports any of these mechanisms as a absolute account of metamorphopsia. Recently, Ooto et al. evaluated the cone anatomy of patients with ERM and axial aqueous chorioretinopathy (CSC) application adaptive eyes scanning laser ophthalmoscopy. Interestingly, a 45% to 70% lower cone anatomy was detected in eyes with bound CSC compared with accustomed eyes,8 but they begin no aberration in beggarly cone anatomy amid the eyes of patients with ERM and accustomed eyes.9 However, disrupted regularity of cone-packing geometry was detected in eyes with ERM, suggesting ataxia of photoreceptor cells.
We speculated that the arrangement of the photoreceptor ataxia would actuate the characteristics of metamorphopsia. Macropsia is a action that affects animal beheld acumen in which altar are perceived to be beyond than they absolutely are. Theoretically, macropsia can advance back the axial cone beef are concentrically displaced from their aboriginal site. Such displacement could be detected by spectral-domain optical adherence tomography (SD-OCT), which has bigger the acceleration and acuteness of OCT, acceptance scanning at a college resolution.10–14
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